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Immuno-Grade GPR25 Receptor Antibodies

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GPR25 (IHC-grade), G Protein-Coupled Receptor 25 Antibody
GPR25 (IHC-grade), G Protein-Coupled Receptor...
The GPR25 antibody is directed against the distal end of the carboxyl-terminal tail of mouse, rat and human GRP25. It can be used to detect total GPR25 receptors in Western blots independent of phosphorylation. The GPR25 antibody can...
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GPR25 (G Protein-Coupled Receptor 25) is an orphan GPCR with no confirmed endogenous ligand or well-defined signaling pathway. Its biological functions remain largely unclear, but genetic studies suggest potential roles in inflammation and cardiovascular regulation. GPR25 shows relatively low and tissue-specific expression, with detectable levels in the vascular system, skeletal muscle, and certain immune cells. Its expression pattern suggests possible involvement in vascular and immune-related processes. While GPR25 is not currently a major focus in drug development, it has attracted interest in genetic association studies, where variants in the GPR25 gene have been linked to blood pressure regulation and inflammatory conditions. For more information on GPR25 pharmacology please refer to the IUPHAR database. For further reading refer to:

Davenport AP, Alexander SP, Sharman JL, Pawson AJ, Benson HE, Monaghan AE, Liew WC, Mpamhanga CP, Bonner TI, Neubig RR, Pin JP, Spedding M, Harmar AJ. International Union of Basic and Clinical Pharmacology. LXXXVIII. G protein-coupled receptor list: recommendations for new pairings with cognate ligands. Pharmacol Rev. 2013 May 17;65(3):967-86. doi: 10.1124/pr.112.007179. PMID: 23686350; PMCID: PMC3698937.

Alexander SP, Battey J, Benson HE, Benya RV, Bonner TI, Davenport AP, Dhanachandra Singh K, Eguchi S, Harmar A, Holliday N, Jensen RT, Karnik S, Kostenis E, Liew WC, Monaghan AE, Mpamhanga C, Neubig R, Pawson AJ, Pin JP, Sharman JL, Spedding M, Spindel E, Stoddart L, Storjohann L, Thomas WG, Tirupula K, Vanderheyden P. Class A Orphans in GtoPdb v.2023.1. IUPHAR/BPS Guide to Pharmacology CITE. 2023; 2023(1). Available from: https://doi.org/10.2218/gtopdb/F16/2023.1.

GPR25 (G Protein-Coupled Receptor 25) is an orphan GPCR with no confirmed endogenous ligand or well-defined signaling pathway. Its biological functions remain largely unclear, but genetic studies... read more »
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Immuno-Grade GPR25 Receptor Antibodies

GPR25 (G Protein-Coupled Receptor 25) is an orphan GPCR with no confirmed endogenous ligand or well-defined signaling pathway. Its biological functions remain largely unclear, but genetic studies suggest potential roles in inflammation and cardiovascular regulation. GPR25 shows relatively low and tissue-specific expression, with detectable levels in the vascular system, skeletal muscle, and certain immune cells. Its expression pattern suggests possible involvement in vascular and immune-related processes. While GPR25 is not currently a major focus in drug development, it has attracted interest in genetic association studies, where variants in the GPR25 gene have been linked to blood pressure regulation and inflammatory conditions. For more information on GPR25 pharmacology please refer to the IUPHAR database. For further reading refer to:

Davenport AP, Alexander SP, Sharman JL, Pawson AJ, Benson HE, Monaghan AE, Liew WC, Mpamhanga CP, Bonner TI, Neubig RR, Pin JP, Spedding M, Harmar AJ. International Union of Basic and Clinical Pharmacology. LXXXVIII. G protein-coupled receptor list: recommendations for new pairings with cognate ligands. Pharmacol Rev. 2013 May 17;65(3):967-86. doi: 10.1124/pr.112.007179. PMID: 23686350; PMCID: PMC3698937.

Alexander SP, Battey J, Benson HE, Benya RV, Bonner TI, Davenport AP, Dhanachandra Singh K, Eguchi S, Harmar A, Holliday N, Jensen RT, Karnik S, Kostenis E, Liew WC, Monaghan AE, Mpamhanga C, Neubig R, Pawson AJ, Pin JP, Sharman JL, Spedding M, Spindel E, Stoddart L, Storjohann L, Thomas WG, Tirupula K, Vanderheyden P. Class A Orphans in GtoPdb v.2023.1. IUPHAR/BPS Guide to Pharmacology CITE. 2023; 2023(1). Available from: https://doi.org/10.2218/gtopdb/F16/2023.1.

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